AKI assessment - How do patients present?
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Not usually a problem unless the patient has a reduced conscious level (a late sign of serious organ failure).
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The patient may present with:
Raised respiratory rate and/ or breathlessness. This can be due to compensation for metabolic acidosis or may be due to fluid overload.
On auscultation: bi-basal fine crackles or reduced breath sounds at the bases if developing fluid overload.
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The patient may present:
hypotensive and tachycardic as a result of hypovolaemia/ shock
Other signs of this will include dry mucous membranes, cool peripheries, delayed CRT, decreased skin turgor
The patient may feel thirsty
Peripheral oedema if developing fluid overload
low urine output, concentrated urine
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The patient may present:
drowsy or with new confusion due to the accumulation of urea or drugs
headaches or lightheadedness due to hypovolaemia
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May have a distended/ palpable bladder if outflow obstruction
May present with abdominal or flank pain if presenting with intrinsic renal pathology